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I can’t hear you very well msn on viagra In a mouse model of MLD, we have demonstrated that disease manifestations can be prevented and corrected by lentiviral vector (LV)–based HSC-GT but not by HSCT (8, 9, 16). This is consistent with the observation that HSCT fails to provide consistent benefits in MLD patients (3, 5–7). LV–based HSC-GT induced extensive and supra-physiological expression of the functional ARSA gene throughout the HSC progeny, which in turn mediated widespread cross-correction of CNS and PNS resident cells (8, 9).